Which Foods are Recommended for Atypical Teratoid Rhabdoid Tumor?

Highlights

No two cancers are the same, nor are they treated the same, and neither should nutrition be the same for everyone. Nutrition includes foods like pulses, vegetables, fruits, nuts, oils, herbs and spices. Also nutrition includes supplements which are high concentrations of foods or high concentrations of individual ingredients found in foods. For cancers like Atypical Teratoid Rhabdoid Tumor when undergoing chemotherapy or when you determine you have a genetic risk for developing Atypical Teratoid Rhabdoid Tumor because of SMARCB1 and SETD2 gene mutations, a very important question is “What foods should I avoid and what foods are recommended specifically for me?”. The other related question is “What nutritional supplements should I avoid?”.

There is no one answer to this question for cancers such as Atypical Teratoid Rhabdoid Tumor which can be found through internet searches. The answer to the question is “It Depends” because the nutrition plan needs to be personalized for you. Nutrition should depend on the cancer indication, genetic information, adult or pediatric, staging, primary or secondary, advanced, metastatic, relapsed or refractory, ongoing treatments if any, nutritional supplements being taken, age and factors like gender, weight, height, lifestyle, allergies and food preferences.

In short – the process to answer questions like “Should I Avoid eating fruit Cranberry” or “Include fruit Persimmon in my diet” or “Should I reduce consumption of vegetable Lotus” or “Can I take Dim and Andrographis supplements” is not as simple as internet searches. The process is very complex and answers are based on knowhow of genetics, action of treatments, active ingredients in foods and their associated biological action. Finally the answer to the nutrition question needs to be personalized for you.

RECOMMENDATION: PERSONALIZE YOUR FOODS AND SUPPLEMENTS TO ATYPICAL TERATOID RHABDOID TUMOR, TREATMENTS, GENETIC INFORMATION, AND OTHER CONDITIONS.

The overall objective of personalized nutrition for Atypical Teratoid Rhabdoid Tumor is to minimize foods and nutritional supplements which have adverse interactions with cancer molecular drivers and ongoing treatments. And identify those foods and supplements which have a beneficial action. Whenever there are changes in treatments or diagnosis – it is important to remember that your foods and supplements need re-evaluation. And the answers to the nutrition question could be different based on the new context.

RECOMMENDATION: UPDATE YOUR NUTRITION FOR ATYPICAL TERATOID RHABDOID TUMOR, WHEN TREATMENTS, DISEASE STATUS AND OTHER CONDITIONS CHANGE.

About Atypical Teratoid Rhabdoid Tumor

cBioPortal is one source of collection of cancer patient data from clinical trials across 350 plus cancer indications. The data from each clinical trial includes the clinical trial name and study details like number of patients, ages, gender, ethnicity, treatments, tumor site, genetic aberrations found and analysis of all the data. The cBioPortal for Cancer Genomics was originally developed at Memorial Sloan Kettering Cancer Center (MSK). The public cBioPortal site is hosted by the Center for Molecular Oncology at MSK – https://www.cbioportal.org/about.

Following key highlights are derived from clinical data for Atypical Teratoid Rhabdoid Tumor from cBioPortal. The patients enrolled in the studies for Atypical Teratoid Rhabdoid Tumor are in ages between 2 to 17 with an average age of 3. 56.6% of males and 43.4% of females were the distribution of gender in these clinical studies. From a patient sample size of 39; the top genes with mutations and other abnormalities for Atypical Teratoid Rhabdoid Tumor include genes SMARCB1, SETD2, CIT, MUC4 and COL6A6. The occurrence frequency distribution for these genes respectively is 17.5%, 7.5%, 7.5%, 7.5% and 5.0%. These tumor genetic details of Atypical Teratoid Rhabdoid Tumor are mapped to molecular biochemical pathway drivers of cancer thereby providing definition of characteristic features of Atypical Teratoid Rhabdoid Tumor.

Atypical teratoid rhabdoid tumor (ATRT) is a primary central nervous system tumor that begins in the brain and spinal cord. They are malignant and fast-growing tumors classified as Grade IV tumors. Most atypical teratoid rhabdoid tumors are caused due to a change (mutation) in a gene known as SMARCB1, that normally signals proteins to stop tumor growth, but in ATRTs, SMARCB1 does not function properly, and tumor growth is uncontrollable. Symptoms of atypical teratoid rhabdoid tumors include morning headaches, vomiting, changes in activity levels, loss of balance and since ATRTs are fast-growing, the symptoms usually get worse quickly. ATRT can occur in both children and adults and are very rare in both groups with an estimated 58 people diagnosed per year and an estimated 596 people living with this tumor (Ref: cancer.gov). The relative 5-year survival rate for ATRT is 32.2% but factors such as tumor grade and type, person’s age and health when diagnosed and how they respond to treatment, can affect the prognosis. Treatment options for atypical teratoid rhabdoid tumors include surgery, radiation, chemotherapy or enrolling in clinical trials with new chemotherapy, targeted therapy or immunotherapy drugs may also be possible option. Treatments supported by the right nutrition (foods and natural supplements) can enhance treatment effectiveness and well-being of the patient. (Ref: https://www.cancer.gov/rare-brain-spine-tumor/tumors/atrt; https://www.cancer.gov/types/brain/hp/child-cns-atrt-treatment-pdq)

Significance of Nutrition for Atypical Teratoid Rhabdoid Tumor

All foods and nutritional supplements consist of a collection of one or more active chemical ingredients in different proportions and quantities. The action of some active ingredients in a food can have adverse interactions while other active ingredients in the same food may be supportive from the context of Atypical Teratoid Rhabdoid Tumor. Hence the same food has good and not-so-good actions and analysis of combined effect will be needed to come up with a personalized nutrition plan.

For example Cranberry includes active ingredients Quercetin, Ellagic Acid, Vitamin C, Hyperoside, Resveratrol and others. And Persimmon contains active ingredients Quercetin, Betulinic Acid, Vitamin C, Lupeol, Lycopene and others. It is likely that some of these active ingredients of the same food could have opposing effects and hence it is recommended to identify recommended foods based on analysis of all high quantity ingredients contained in foods.

For cancers like Atypical Teratoid Rhabdoid Tumor, activation or inhibition of selected biochemical pathways like DNA Repair, Hypoxia, MAPK Signaling, Post Translation Modification plays an important role in driving cancer growth. Similarly different treatments work via different molecular actions which should never be canceled out by your foods and supplements. The foods and nutritional supplements contain different active ingredients each of which have a specific molecular action on different biochemical pathways. Hence, eating some foods and nutritional supplements would be recommended with a specific treatment of Atypical Teratoid Rhabdoid Tumor, while eating some other foods and supplements may not be recommended.

One common mistake when finding foods to eat or not – is to consider only a few active ingredients contained in foods based on internet searches and ignore the rest. Because different active ingredients contained in foods may have opposing effects on relevant biochemical pathways – it is recommended to consider all the high quantity active ingredients that are present in significant and much larger than trace amounts in the food.

RECOMMENDATION: TO FIND RECOMMENDED AND NON-RECOMMENDED FOODS FOR ATYPICAL TERATOID RHABDOID TUMOR – CONSIDER HIGH QUANTITY ACTIVE INGREDIENTS CONTAINED IN FOODS.

Foods for Atypical Teratoid Rhabdoid Tumor undergoing chemotherapy treatment

In Atypical Teratoid Rhabdoid Tumor – the genes SMARCB1, SETD2, CIT, MUC4 and COL6A6 have high occurrences of genomic abnormalities. Not all of these genes necessarily are relevant for cancer – though they have been reported. Some of these genes directly or indirectly end up manipulating different cancer related biochemical biological pathways. Some of the pathways which are relevant drivers for Atypical Teratoid Rhabdoid Tumor are DNA Repair, Hypoxia, MAPK Signaling and others. Temozolomide is one of the chemotherapies used for cancer treatment. The intent of treatment is to negate or cancel out effects of biochemical pathway drivers DNA Repair, Hypoxia, MAPK Signaling so as to reduce disease progression and inhibit growth. Those foods whose combined action of active ingredients support treatment action and do not enhance disease drivers are recommended foods and supplements which will be included in personalized nutrition. And similarly – those foods whose combined action of active ingredients is not supportive of treatment action but end up promoting disease drives will not be recommended in your personalized nutrition plan.

RECOMMENDATION: AVOID SUPPLEMENTS AND FOODS WHICH ARE NOT SUPPORTIVE OF CANCER TREATMENT ACTION AND RATHER ENHANCE DISEASE DRIVERS.

Eat more pulses, Common Pea or Moth Bean?

Pulses are an important part of many diets. The active ingredients contained in Common Pea are Vitamin C, Genistein, Lupeol, Daidzein, Beta-sitosterol among others. While the active ingredients contained in Moth Bean are Beta-sitosterol, Linolenic Acid, Stigmasterol, Oleic Acid, Vitamin A and others.

Vitamin C can manipulate biochemical pathways Epithelial to Mesenchymal Transition, Adherens junction and Growth Factor Signaling. Beta-sitosterol has biological action on biochemical pathways Microtubule Dynamics, DNA Repair and Cell Cycle.

Vitamin A can manipulate biochemical pathways Focal Adhesion. Linolenic Acid has biological action on biochemical pathways Oxidative Stress. And so on.

When treating Atypical Teratoid Rhabdoid Tumor with chemotherapy Temozolomide – Foods like Common Pea are recommended compared to Moth Bean. This is because the active ingredients Vitamin A and Linolenic Acid in Moth Bean interferes with treatment action by canceling out the biochemical pathways through which the chemotherapy works. While the active ingredients Vitamin C and Beta-sitosterol contained in Common Pea support the treatment action by enhancing the biochemical pathway effect through which the chemotherapy works.

RECOMMENDATION: COMMON PEA IS RECOMMENDED OVER MOTH BEAN FOR ATYPICAL TERATOID RHABDOID TUMOR ON TREATMENT WITH CHEMOTHERAPY TEMOZOLOMIDE FOR SOME CONDITIONS.

Eat more vegetables, Cassava or Lotus?

Vegetables are an important part of many diets. The active ingredients contained in Cassava are Vitamin C, Beta-sitosterol, Linolenic Acid, Oleic Acid, Vitamin A among others. While the active ingredients contained in Lotus are Quercetin, Vitamin C, Beta-sitosterol, Linolenic Acid, Fisetin and others.

Vitamin C can manipulate biochemical pathways Epithelial to Mesenchymal Transition, Adherens junction and Growth Factor Signaling. Beta-sitosterol has biological action on biochemical pathways Microtubule Dynamics, DNA Repair and Cell Cycle.

Citric Acid can manipulate biochemical pathways Oxidative Stress. Quercetin has biological action on biochemical pathways Oxidative Stress. And so on.

When treating Atypical Teratoid Rhabdoid Tumor with chemotherapy Temozolomide – Foods like Cassava are recommended compared to Lotus. This is because the active ingredients Citric Acid and Quercetin in Lotus interferes with treatment action by canceling out the biochemical pathways through which the chemotherapy works. While the active ingredients Vitamin C and Beta-sitosterol contained in Cassava support the treatment action by enhancing the biochemical pathway effect through which the chemotherapy works.

RECOMMENDATION: CASSAVA IS RECOMMENDED OVER LOTUS FOR ATYPICAL TERATOID RHABDOID TUMOR ON TREATMENT WITH CHEMOTHERAPY TEMOZOLOMIDE FOR SOME CONDITIONS.

Eat more fruits, Persimmon or Cranberry?

Fruits are an important part of many diets. The active ingredients contained in Persimmon are Quercetin, Betulinic Acid, Vitamin C, Lupeol, Lycopene among others. While the active ingredients contained in Cranberry are Quercetin, Ellagic Acid, Vitamin C, Hyperoside, Resveratrol and others.

Vitamin C can manipulate biochemical pathways Epithelial to Mesenchymal Transition, Adherens junction and Growth Factor Signaling. Lycopene has biological action on biochemical pathways Microtubule Dynamics, JAK-STAT Signaling and Cell Cycle.

Quercetin can manipulate biochemical pathways Oxidative Stress. Ellagic Acid has biological action on biochemical pathways Epithelial to Mesenchymal Transition, Stem Cell Signaling and Microtubule Dynamics. And so on.

When treating Atypical Teratoid Rhabdoid Tumor with chemotherapy Temozolomide – Foods like Persimmon are recommended compared to Cranberry. This is because the active ingredients Quercetin and Ellagic Acid in Cranberry interferes with treatment action by canceling out the biochemical pathways through which the chemotherapy works. While the active ingredients Vitamin C and Lycopene contained in Persimmon support the treatment action by enhancing the biochemical pathway effect through which the chemotherapy works.

RECOMMENDATION: PERSIMMON IS RECOMMENDED OVER CRANBERRY FOR ATYPICAL TERATOID RHABDOID TUMOR ON TREATMENT WITH CHEMOTHERAPY TEMOZOLOMIDE FOR SOME CONDITIONS.

Eat more nuts, Pine Nut or Pumpkin Seeds?

Nuts are an important part of many diets. The active ingredients contained in Pine Nut are Vitamin E, Beta-sitosterol, Linolenic Acid, Oleic Acid, Vitamin A among others. While the active ingredients contained in Pumpkin Seeds are Beta-sitosterol, Salicylic Acid, Linolenic Acid, Stigmasterol, Vitamin B3 and others.

Beta-sitosterol can manipulate biochemical pathways Epithelial to Mesenchymal Transition, Adherens junction and Microtubule Dynamics. Vitamin K has biological action on biochemical pathways MYC Signaling, Cell Cycle and PI3K-AKT-MTOR Signaling.

Gamma-linolenic Acid can manipulate biochemical pathways Oxidative Stress and Hypoxia. Lecithin has biological action on biochemical pathways JAK-STAT Signaling, MYC Signaling and MAPK Signaling. And so on.

When treating Atypical Teratoid Rhabdoid Tumor with chemotherapy Temozolomide – Foods like Pine Nut are recommended compared to Pumpkin Seeds. This is because the active ingredients Gamma-linolenic Acid and Lecithin in Pumpkin Seeds interferes with treatment action by canceling out the biochemical pathways through which the chemotherapy works. While the active ingredients Beta-sitosterol and Vitamin K contained in Pine Nut support the treatment action by enhancing the biochemical pathway effect through which the chemotherapy works.

RECOMMENDATION: PINE NUT IS RECOMMENDED OVER PUMPKIN SEEDS FOR ATYPICAL TERATOID RHABDOID TUMOR ON TREATMENT WITH CHEMOTHERAPY TEMOZOLOMIDE FOR SOME CONDITIONS.

Foods for Genetic Risk of Atypical Teratoid Rhabdoid Tumor

One of the ways to assess risk of cancer is by checking for presence of genetic abnormalities in a set of genes. There is prior information on a list of genes whose mutations and other aberrations can play a role in risk to different cancers. SMARCB1 and SETD2 are two genes whose abnormalities are risk factors for Atypical Teratoid Rhabdoid Tumor. In such a cancer risk situation – while there are typically no treatments which a physician can prescribe – the various biochemical pathways which are potentially molecular drivers of Atypical Teratoid Rhabdoid Tumor can be used as a guide for coming up with a recommended personalized nutrition plan. For Atypical Teratoid Rhabdoid Tumor gene SMARCB1 has causative impact on biological pathways like Androgen Signaling, Chromatin Remodeling and Epithelial to Mesenchymal Transition. And SETD2 has a causative impact on biological pathways like Oncogenic Histone Methylation. Foods and nutritional supplements which have molecular action to cancel out biochemical pathways effects of genes like SMARCB1 and SETD2 should be included in a personalized nutrition plan. And those foods and supplements which promote the effects of genes SMARCB1 and SETD2 should be avoided.

Eat more pulses, Scarlet Bean or Pigeon Pea?

The active ingredients contained in Scarlet Bean are Beta-sitosterol, Vitamin C, Oleic Acid, Genistein, Linolenic Acid among others. While the active ingredients contained in Pigeon Pea are Vitamin C, Oleic Acid, Genistein, Linolenic Acid, Linoleic Acid and others.

Beta-sitosterol can manipulate biochemical pathways Apoptosis, MYC Signaling and P53 Signaling. Vitamin C has biological action on biochemical pathways Growth Factor Signaling, Cell Cycle Checkpoints and PI3K-AKT-MTOR Signaling.

Linoleic Acid can manipulate biochemical pathways Growth Factor Signaling. Folic Acid has biological action on biochemical pathways Apoptosis, MYC Signaling and P53 Signaling. And so on.

For genetic risk of Atypical Teratoid Rhabdoid Tumor due to abnormalities in genes SMARCB1 and SETD2 – Foods like Scarlet Bean are recommended compared to Pigeon Pea. This is because the active ingredients Linoleic Acid and Folic Acid in Pigeon Pea further promote the effects of genes on the biochemical pathways. While the active ingredients Beta-sitosterol and Vitamin C contained in Scarlet Bean together have a canceling effect of genes on the biochemical pathways.

RECOMMENDATION: SCARLET BEAN IS RECOMMENDED OVER PIGEON PEA FOR REDUCING THE GENETIC RISK OF ATYPICAL TERATOID RHABDOID TUMOR DUE TO GENES SMARCB1 AND SETD2

Eat more vegetables, Jicama or Radish?

The active ingredients contained in Jicama are Vitamin C, Beta-carotene, Vitamin B3, Vitamin A, Folic Acid among others. While the active ingredients contained in Radish are Quercetin, Beta-sitosterol, Vitamin C, Oleic Acid, P-coumaric Acid and others.

Vitamin C can manipulate biochemical pathways Apoptosis, Growth Factor Signaling and MYC Signaling. Beta-carotene has biological action on biochemical pathways P53 Signaling, Cell Cycle Checkpoints and PI3K-AKT-MTOR Signaling.

Pelargonidin can manipulate biochemical pathways MYC Signaling, Cell Cycle Checkpoints and PI3K-AKT-MTOR Signaling. Linoleic Acid has biological action on biochemical pathways Growth Factor Signaling. And so on.

For genetic risk of Atypical Teratoid Rhabdoid Tumor due to abnormalities in genes SMARCB1 and SETD2 – Foods like Jicama are recommended compared to Radish. This is because the active ingredients Pelargonidin and Linoleic Acid in Radish further promote the effects of genes on the biochemical pathways. While the active ingredients Vitamin C and Beta-carotene contained in Jicama together have a canceling effect of genes on the biochemical pathways.

RECOMMENDATION: JICAMA IS RECOMMENDED OVER RADISH FOR REDUCING THE GENETIC RISK OF ATYPICAL TERATOID RHABDOID TUMOR DUE TO GENES SMARCB1 AND SETD2

Eat more fruits, Feijoa or Lingonberry?

The active ingredients contained in Feijoa are Lycopene, Vitamin C, Casuarinin, Folic Acid among others. While the active ingredients contained in Lingonberry are Quercetin, Hyperoside, Resveratrol, Vitamin C, Oleic Acid and others.

Vitamin C can manipulate biochemical pathways Apoptosis, Growth Factor Signaling and MYC Signaling. Lycopene has biological action on biochemical pathways P53 Signaling, Cell Cycle Checkpoints and PI3K-AKT-MTOR Signaling.

Resveratrol can manipulate biochemical pathways P53 Signaling. Linoleic Acid has biological action on biochemical pathways Growth Factor Signaling. And so on.

For genetic risk of Atypical Teratoid Rhabdoid Tumor due to abnormalities in genes SMARCB1 and SETD2 – Foods like Feijoa are recommended compared to Lingonberry. This is because the active ingredients Resveratrol and Linoleic Acid in Lingonberry further promote the effects of genes on the biochemical pathways. While the active ingredients Vitamin C and Lycopene contained in Feijoa together have a canceling effect of genes on the biochemical pathways.

RECOMMENDATION: FEIJOA IS RECOMMENDED OVER LINGONBERRY FOR REDUCING THE GENETIC RISK OF ATYPICAL TERATOID RHABDOID TUMOR DUE TO GENES SMARCB1 AND SETD2

Eat more nuts, Pecan Nut or Brazil Nut?

The active ingredients contained in Pecan Nut are Vitamin E, Delphinidin, Cianidanol, Oleic Acid, Linolenic Acid among others. While the active ingredients contained in Brazil Nut are Vitamin E, Oleic Acid, Linolenic Acid, Lecithin, Folic Acid and others.

Vitamin E can manipulate biochemical pathways Apoptosis, Growth Factor Signaling and MYC Signaling. Delphinidin has biological action on biochemical pathways RAS-RAF Signaling, PI3K-AKT-MTOR Signaling and MYC Signaling.

Lecithin can manipulate biochemical pathways MYC Signaling and PI3K-AKT-MTOR Signaling. Folic Acid has biological action on biochemical pathways Apoptosis, P53 Signaling and Cell Cycle Checkpoints. And so on.

For genetic risk of Atypical Teratoid Rhabdoid Tumor due to abnormalities in genes SMARCB1 and SETD2 – Foods like Pecan Nut are recommended compared to Brazil Nut. This is because the active ingredients Lecithin and Folic Acid in Brazil Nut further promote the effects of genes on the biochemical pathways. While the active ingredients Vitamin E and Delphinidin contained in Pecan Nut together have a canceling effect of genes on the biochemical pathways.

RECOMMENDATION: PECAN NUT IS RECOMMENDED OVER BRAZIL NUT FOR REDUCING THE GENETIC RISK OF ATYPICAL TERATOID RHABDOID TUMOR DUE TO GENES SMARCB1 AND SETD2

In Summary

An important thing to remember is that cancer treatments may not be the same for everyone – and neither should your nutrition be. Nutrition which includes food and nutritional supplements is a very effective tool controlled by you.

“What should I eat?” is the most frequently asked question in the context of cancer. The answer calculation is complex and depends upon cancer type, underlying genomics, current treatments, any allergies, lifestyle information, and factors like BMI.

The addon personalized nutrition plan recommends foods and supplements which minimizes adverse nutrition interactions and encourages support to treatments.

You can get started NOW and design a personalized nutrition plan for Atypical Teratoid Rhabdoid Tumor by answering questions on type of cancer, current treatments, supplements, allergies, age group, gender, and lifestyle information.

What food you eat and which supplements you take is a decision you make. Your decision should include consideration of the cancer gene mutations, which cancer, ongoing treatments and supplements, any allergies, lifestyle information, weight, height and habits.

The nutrition planning for cancer from addon is not based on internet searches. It automates the decision making for you based on molecular science implemented by our scientists and software engineers. Irrespective of whether you care to understand the underlying biochemical molecular pathways or not - for nutrition planning for cancer that understanding is needed.

Get started NOW with your nutrition planning by answering questions on the name of cancer, genetic mutations, ongoing treatments and supplements, any allergies, habits, lifestyle, age group and gender.

References

• Brain Cptac 2020
• Integrated Proteogenomic Characterization across Major Histological Types of Pediatric Brain Cancer.
• Vitamin C enhances epigenetic modifications induced by 5-azacytidine and cell cycle arrest in the hepatocellular carcinoma cell lines HLE and Huh7.
• β-Sitosterol targets Trx/Trx1 reductase to induce apoptosis in A549 cells via ROS mediated mitochondrial dysregulation and p53 activation.
• Adhesion to the extracellular matrix is positively regulated by retinoic acid in HepG2 cells.
• Alpha-linolenic acid confers protection on mice renal cells against cisplatin-induced nephrotoxicity.
• Lycopene metabolite, apo-10′-lycopenoic acid, inhibits diethylnitrosamine-initiated, high fat diet-promoted hepatic inflammation and tumorigenesis in mice.
• Small-molecule inhibitors of NADPH oxidase 4.
• Identification of ellagic acid as potent inhibitor of protein kinase CK2: a successful example of a virtual screening application.
• Research progress on the anticancer effects of vitamin K2.
• Research on Rett syndrome: strategy and preliminary results.
• The recruitment of Raf-1 to membranes is mediated by direct interaction with phosphatidic acid and is independent of association with Ras.
• Role of phospholipase D in migration and invasion induced by linoleic acid in breast cancer cells.
• Effects of folate deficiency on gene expression in the apoptosis and cancer pathways in colon cancer cells.
• Resveratrol, a remarkable inhibitor of ribonucleotide reductase.
• The effect of Pasteurella haemolytica cytotoxin on bovine polymorphonuclear leukocytes can be attenuated by beta-adrenoceptor antagonists.
• Pelargonidin suppresses adipogenesis in 3T3-L1 cells through inhibition of PPAR-γ signaling pathway.
• Gamma- and delta-tocotrienols exert a more potent anticancer effect than alpha-tocopheryl succinate on breast cancer cell lines irrespective of HER-2/neu expression.
• Identification of new potent phthalazine derivatives with VEGFR-2 and EGFR kinase inhibitory activity.
• HyperFoods: Machine intelligent mapping of cancer-beating molecules in foods.

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